mouse anti osa Search Results


mouse anti osa  (Developmental Studies Hybridoma Bank)
93
Developmental Studies Hybridoma Bank mouse anti osa
Mouse Anti Osa, supplied by Developmental Studies Hybridoma Bank, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse anti osa/product/Developmental Studies Hybridoma Bank
Average 93 stars, based on 1 article reviews
mouse anti osa - by Bioz Stars, 2026-06
93/100 stars
  Buy from Supplier
mouse monoclonal anti-ho-1  (Enzo Biochem)
90
Enzo Biochem mouse monoclonal anti-ho-1
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Mouse Monoclonal Anti Ho 1, supplied by Enzo Biochem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/mouse monoclonal anti-ho-1/product/Enzo Biochem
Average 90 stars, based on 1 article reviews
mouse monoclonal anti-ho-1 - by Bioz Stars, 2026-06
90/100 stars
  Buy from Supplier
anti‑mouse heme oxygenase‑1 (ho‑1) (adi‑osa‑110) antibody  (Enzo Biochem)
90
Enzo Biochem anti‑mouse heme oxygenase‑1 (ho‑1) (adi‑osa‑110) antibody
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Anti‑Mouse Heme Oxygenase‑1 (Ho‑1) (Adi‑Osa‑110) Antibody, supplied by Enzo Biochem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti‑mouse heme oxygenase‑1 (ho‑1) (adi‑osa‑110) antibody/product/Enzo Biochem
Average 90 stars, based on 1 article reviews
anti‑mouse heme oxygenase‑1 (ho‑1) (adi‑osa‑110) antibody - by Bioz Stars, 2026-06
90/100 stars
  Buy from Supplier
ho 1  (Cell Signaling Technology Inc)
96
Cell Signaling Technology Inc ho 1
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Ho 1, supplied by Cell Signaling Technology Inc, used in various techniques. Bioz Stars score: 96/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/ho 1/product/Cell Signaling Technology Inc
Average 96 stars, based on 1 article reviews
ho 1 - by Bioz Stars, 2026-06
96/100 stars
  Buy from Supplier
anti ho 1 osa 111 monoclonal antibodies  (Bio-Rad)
93
Bio-Rad anti ho 1 osa 111 monoclonal antibodies
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Anti Ho 1 Osa 111 Monoclonal Antibodies, supplied by Bio-Rad, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti ho 1 osa 111 monoclonal antibodies/product/Bio-Rad
Average 93 stars, based on 1 article reviews
anti ho 1 osa 111 monoclonal antibodies - by Bioz Stars, 2026-06
93/100 stars
  Buy from Supplier
anti-heme oxygenase-1 (ho-1; #adi-osa-110)  (Enzo Biochem)
90
Enzo Biochem anti-heme oxygenase-1 (ho-1; #adi-osa-110)
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Anti Heme Oxygenase 1 (Ho 1; #Adi Osa 110), supplied by Enzo Biochem, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti-heme oxygenase-1 (ho-1; #adi-osa-110)/product/Enzo Biochem
Average 90 stars, based on 1 article reviews
anti-heme oxygenase-1 (ho-1; #adi-osa-110) - by Bioz Stars, 2026-06
90/100 stars
  Buy from Supplier
ho 1 mouse monoclonal igg2b  (Stressgen Biotechnologies)
86
Stressgen Biotechnologies ho 1 mouse monoclonal igg2b
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Ho 1 Mouse Monoclonal Igg2b, supplied by Stressgen Biotechnologies, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/ho 1 mouse monoclonal igg2b/product/Stressgen Biotechnologies
Average 86 stars, based on 1 article reviews
ho 1 mouse monoclonal igg2b - by Bioz Stars, 2026-06
86/100 stars
  Buy from Supplier
anti baf250 osa 1 2  (Bethyl)
93
Bethyl anti baf250 osa 1 2
miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 <t>(HO-1).</t> ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.
Anti Baf250 Osa 1 2, supplied by Bethyl, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/result/anti baf250 osa 1 2/product/Bethyl
Average 93 stars, based on 1 article reviews
anti baf250 osa 1 2 - by Bioz Stars, 2026-06
93/100 stars
  Buy from Supplier
human n acetylglucosaminyltransferase v mgat5 antibody  (R&D Systems)
N/A
The Human N Acetylglucosaminyltransferase V MGAT5 Antibody from R D Systems is a mouse monoclonal antibody to N Acetylglucosaminyltransferase V MGAT5 This antibody reacts with human The Human N Acetylglucosaminyltransferase V MGAT5 Antibody has been
   Buy from Supplier
n sulfoglucosamine sulfohydrolase antibody  (Abbexa)
N/A
N Sulfoglucosamine Sulfohydrolase Antibody is a Rabbit Polyclonal antibody against N Sulfoglucosamine Sulfohydrolase
   Buy from Supplier
st6 beta galactosamide alpha 2 6 sialyltranferase 1 antibody  (Abbexa)
N/A
St6 Beta Galactosamide Alpha 2 6 Sialyltranferase 1 Antibody is a Rabbit Polyclonal antibody against St6 Beta Galactosamide Alpha 2 6 Sialyltranferase 1
   Buy from Supplier
mouse anti human cd3:fitc/hla-class 2:rpe dual color reagent (50tests)  (Aviva Systems)
N/A
MOUSE ANTI HUMAN CD3:FITC/HLA-CLASS 2:RPE; MOUSE ANTI HUMAN CD3:FITC/HLA-CLASS 2:RPE_x000D_
   Buy from Supplier

Image Search Results


miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 (HO-1). ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.

Journal: Aging (Albany NY)

Article Title: miR-183-5p alleviates early injury after intracerebral hemorrhage by inhibiting heme oxygenase-1 expression

doi: 10.18632/aging.103343

Figure Lengend Snippet: miR-183-5p alleviated early inflammation and oxidative damage by directly targeting heme oxygenase-1 (HO-1). ( A ) Above: schematic showing the potential miR-183-5p binding site in the HO-1 3’-untranslated region (3’-UTR). A mutant (MUT) HO-1 3’-UTR was introduced by replacing the wild type (WT) binding sequence with a mutant sequence. Below: inhibition of relative luciferase activity of HO-1 3’-UTR reporter molecules in human embryonic kidney 293 cells mediated by miR-183-5p. miR-183-5p MM, miR-183-5p mimic; NC, nontarget control. ( B ) Above: western blotting revealed that miRNA-183-5p downregulated HO-1 expression. Below: quantitative analysis of HO-1 protein expression in different groups. n = 8/group. ( C ) Quantitative analysis of cytokine expression in the brains of mice from different groups pretreated with the HO-1 inhibitor zinc protoporphyrin IX (ZnPP) at 3 days after ICH. n = 8/group. ( D ) Quantitative analysis of 4-HNE expression in the brains of mice from different groups pretreated with the HO-1 inhibitor ZnPP at 3 days after ICH. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the ICH group.

Article Snippet: The following primary antibodies were used: mouse monoclonal anti-HO-1 (1:100, ADI-OSA-110, Enzo Life Sciences, Inc., Farmingdale, NY, USA), rabbit anti-Iba-1 (1:200, ab178846, Abcam Plc., Cambridge, UK), and rabbit anti-myeloperoxidase (MPO, 1:100, ab9535, Abcam).

Techniques: Binding Assay, Mutagenesis, Sequencing, Inhibition, Luciferase, Activity Assay, Western Blot, Expressing, Standard Deviation

miRNA-183-5p affected microglial survival by targeting heme oxygenase-1 (HO-1) after intracerebral hemorrhage (ICH). ( A ) Left: representative immunofluorescence images of HO-1 in Iba-1–positive microglia at 3 days after ICH. Right: percentage of both Iba-1– and HO-1–positive cells in Iba-1–positive microglia. Scale bars = 50 μm, n = 8/group. * P < 0.05 vs. the ICH group. ( B ) Above: representative immunofluorescence images of HO-1 in BV2 microglia from different groups at 24 hours after hemin treatment. Below: percentage of HO-1–positive BV2 microglia. Scale bars = 50 μm, n = 3/group. * P < 0.05 vs. the hemin group. ZnPP, zinc protoporphyrin IX; CoPP, cobalt protoporphyrin IX.

Journal: Aging (Albany NY)

Article Title: miR-183-5p alleviates early injury after intracerebral hemorrhage by inhibiting heme oxygenase-1 expression

doi: 10.18632/aging.103343

Figure Lengend Snippet: miRNA-183-5p affected microglial survival by targeting heme oxygenase-1 (HO-1) after intracerebral hemorrhage (ICH). ( A ) Left: representative immunofluorescence images of HO-1 in Iba-1–positive microglia at 3 days after ICH. Right: percentage of both Iba-1– and HO-1–positive cells in Iba-1–positive microglia. Scale bars = 50 μm, n = 8/group. * P < 0.05 vs. the ICH group. ( B ) Above: representative immunofluorescence images of HO-1 in BV2 microglia from different groups at 24 hours after hemin treatment. Below: percentage of HO-1–positive BV2 microglia. Scale bars = 50 μm, n = 3/group. * P < 0.05 vs. the hemin group. ZnPP, zinc protoporphyrin IX; CoPP, cobalt protoporphyrin IX.

Article Snippet: The following primary antibodies were used: mouse monoclonal anti-HO-1 (1:100, ADI-OSA-110, Enzo Life Sciences, Inc., Farmingdale, NY, USA), rabbit anti-Iba-1 (1:200, ab178846, Abcam Plc., Cambridge, UK), and rabbit anti-myeloperoxidase (MPO, 1:100, ab9535, Abcam).

Techniques: Immunofluorescence

miR-183-5p regulated heme oxygenase-1 (HO-1) independent of Nrf2. ( A ) Western blotting revealed that miRNA-183-5p is an HO-1–dependent inhibitor of Nrf2 phosphorylation. n = 8/group. ( B ) Quantitative analysis of the relative expression of p-Nrf2 protein in ( A ). ( C ) Quantitative analysis of the HO-1–dependent inhibitory effect of Nrf2 on miR-183-5p by RT-qPCR. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the intracerebral hemorrhaging (ICH) group. ZnPP, zinc protoporphyrin IX; tBHQ, tert-butylhydroquinone.

Journal: Aging (Albany NY)

Article Title: miR-183-5p alleviates early injury after intracerebral hemorrhage by inhibiting heme oxygenase-1 expression

doi: 10.18632/aging.103343

Figure Lengend Snippet: miR-183-5p regulated heme oxygenase-1 (HO-1) independent of Nrf2. ( A ) Western blotting revealed that miRNA-183-5p is an HO-1–dependent inhibitor of Nrf2 phosphorylation. n = 8/group. ( B ) Quantitative analysis of the relative expression of p-Nrf2 protein in ( A ). ( C ) Quantitative analysis of the HO-1–dependent inhibitory effect of Nrf2 on miR-183-5p by RT-qPCR. n = 8/group. Values are presented as the mean ± standard deviation. * P < 0.05 vs. the intracerebral hemorrhaging (ICH) group. ZnPP, zinc protoporphyrin IX; tBHQ, tert-butylhydroquinone.

Article Snippet: The following primary antibodies were used: mouse monoclonal anti-HO-1 (1:100, ADI-OSA-110, Enzo Life Sciences, Inc., Farmingdale, NY, USA), rabbit anti-Iba-1 (1:200, ab178846, Abcam Plc., Cambridge, UK), and rabbit anti-myeloperoxidase (MPO, 1:100, ab9535, Abcam).

Techniques: Western Blot, Expressing, Quantitative RT-PCR, Standard Deviation

The experimental design. Sham group, sham operation group; ICH group, collagenase-induced intracerebral hemorrhage group; miRNA-seq, miRNA sequencing; miR-183-5p, microRNA-183-5p; HO-1, heme oxygenase-1; ICH 1 d, 3 d, 7 d, 14 d, 28 d groups, 1 day, 3 days, 7 days, 14 days, 28 days after collagenase-induced intracerebral hemorrhage groups; ZnPP, HO-1 inhibitor zinc protoporphyrin IX; Nrf2 -/- , nuclear factor erythroid 2-related factor knockout; CoPP, HO-1 inducer cobalt protoporphyrin IX; tBHQ, Nrf2 activator tert-butylhydroquinone.

Journal: Aging (Albany NY)

Article Title: miR-183-5p alleviates early injury after intracerebral hemorrhage by inhibiting heme oxygenase-1 expression

doi: 10.18632/aging.103343

Figure Lengend Snippet: The experimental design. Sham group, sham operation group; ICH group, collagenase-induced intracerebral hemorrhage group; miRNA-seq, miRNA sequencing; miR-183-5p, microRNA-183-5p; HO-1, heme oxygenase-1; ICH 1 d, 3 d, 7 d, 14 d, 28 d groups, 1 day, 3 days, 7 days, 14 days, 28 days after collagenase-induced intracerebral hemorrhage groups; ZnPP, HO-1 inhibitor zinc protoporphyrin IX; Nrf2 -/- , nuclear factor erythroid 2-related factor knockout; CoPP, HO-1 inducer cobalt protoporphyrin IX; tBHQ, Nrf2 activator tert-butylhydroquinone.

Article Snippet: The following primary antibodies were used: mouse monoclonal anti-HO-1 (1:100, ADI-OSA-110, Enzo Life Sciences, Inc., Farmingdale, NY, USA), rabbit anti-Iba-1 (1:200, ab178846, Abcam Plc., Cambridge, UK), and rabbit anti-myeloperoxidase (MPO, 1:100, ab9535, Abcam).

Techniques: Sequencing, Knock-Out